The uterus hypertrophy, is the uterus even increase, muscle thickness of more than 2.5cm above, accompanied by varying degree of bleeding of a disease.
More maternal, menstruation and the uterus is mostly an increased, normal or thickened endometrium, the individual was polypoid, but the pathological examination the majority of normal, a few show hyperplasia, may be diagnosed with uterine hypertrophy. It should be noted with the identification of uterine fibroids, particularly intramural or single muscle nuclear submucosal, their uniform increases Palace is often difficult to identify with uterine hypertrophy, uterine cavity by curettage and B-ultrasound probe to help diagnose. However, there are still a few cases, there can only diagnosed at laparotomy when.
In addition, attention should be paid with adenomyosis, endometrial cancer and other diseases identified.
Chinese medicine treatment can control menorrhagia, and improvement of general condition; male hormone therapy can reduce the amount of bleeding. Conservative treatment is invalid who may wish to consider hysterectomy. 50 years of age with normal ovaries should be retained.
(A) Multi-maternal chronic uterine involution failure: Multi-maternal myometrium between the elastic fibrous tissue and blood vessels surrounding the proliferation of smooth muscle, causing uterine hypertrophy.
(B) of ovarian dysfunction: Estrogen continuous stimulation, can myometrial hypertrophy. Dysfunctional uterine bleeding is a common clinical patients, especially those longer course, there are different levels of the uterus increases.
(C) inflammation: slow annex inflammation, pelvic inflammation and uterine connective tissue of chronic myositis, causing proliferation of collagen fibers within the uterine muscle, so that uterine fibrosis.
(D) of pelvic congestion, causing uterine connective tissue hyperplasia, can also be caused by uterine hypertrophy.
(E) myometrium hardening of the arteries: primary uterine vascular lesions, etc..
Pathological changes 【】
The basic pathological changes of this disease is the myometrium smooth muscle cells and vessel wall changes.
(A) in general, see: the uterus was uniformly increased, muscle hypertrophy up to 2.5 ~ 3.2cm. Off-white or pink cut surface, the hardness increased, braided fiber bundle was arranged. Outer 1 / 3 of muscle blood vessels bulge, normal or intimal thickening, and sometimes can be seen the merger of small smooth muscle tumors (diameter less than 1cm), or endometrial polyps.
(B) microscopy: Image findings are inconsistent, there are several forms: ① pure smooth muscle cell hypertrophy. Microscopic observation and normal myometrium the same, no collagen fibers hyperplasia, there is no significant change in vessel walls; ② collagen fibers within the uterine muscle hyperplasia, uterine fibrosis formation; ③ muscle within the vessel wall changes: arterio-venous significant expansion in the new blood vessels around the Youcheng Mission fibroelastosis.
【Clinical manifestations 】
The main symptoms are excessive menstrual flow, continuing to extend the number of days; also the performance cycle shortened to about 20 days and continuing through the amount of no significant change in the number of days; or expressed as menstrual period extended, but limited by the amount.
Patients, mostly by mothers, and the majority of the above three products. Ill a long time and who was bleeding more than the amount of anemia Maung gynecological examination the uterus even increase, usually 6 weeks of pregnancy the size of a small number of more than eight weeks of pregnancy the size, texture more tenacious. Bilateral ovaries can be slightly increased, there are multiple follicular cysts.
There are many reasons due to disease, and some can prevent its occurrence, such as family planning, prevention of postpartum infection, postpartum uterine contraction should provide timely adverse drug uterine contraction. Note that the appropriate post-natal prone or knee-chest position and post-natal exercise and pour the uterus to prevent and reduce pelvic congestion. Active treatment of ovarian dysfunction, to prevent the continued estrogen stimulation.