Senile vaginitis, also known as atrophic vaginitis; is a non-specific vaginitis. Mostly occurs in women after menopause, but after bilateral oophorectomy or lactating women may also occur.
The main symptoms are increased vaginal discharge, watery or purulent yellow, smelly, severe infections may occur drip vaginal bleeding, and vaginal pain and burning sensation of falling. Such as involving the vestibular mucosa and around the urethra, often frequent urination, dysuria.
According to age, medical history, combined with local genital examination showed flushing, moist vaginal wall hyperemia, with scattered bleeding points, after fornix and cervical most obvious. After stripping the vaginal mucosa can form ulcers. Generally difficult to diagnose. After the formation of chronic inflammation that can occur two results: First vaginal submucosal connective tissue fibrosis, vagina loses its elasticity, the final form of vaginal stenosis and scarring; another case of the vaginal wall adhesion formation vaginal atresia, even more than in the closed form Vaginal empyema. Corporations such cases rare, but serious condition.
And identification of specific vaginitis, vaginal examination should be taken except trichomoniasis, mold and other pathogens. Bloody vaginal discharge and uterine cancer should be identified. Gynecological examination attention uterine size and shape, the source of bleeding and vaginal cytology, if necessary, such as cervical or endometrial biopsy.
Principles of treatment of senile vaginitis, is to enhance the resistance of the vaginal mucosa and inhibit bacterial growth and reproduction. Systemic administration, oral diethylstilbestrol 0.25 ~ 0.5mg, once daily for 7 days. Sometimes medication can cause withdrawal bleeding. Local administration, improve vaginal pH, with 1% lactic acid or potassium permanganate 1:5000 douching. Night vaginal suppository can put the ethanol film 0.25 ~ 0.5mg, into the vagina once a day for 7 days. If necessary, spreading locally applied antibiotic powder or ointment.
The main reason is due to ovarian failure, low estrogen levels or lack of vaginal epithelial cells decrease in glycogen, the vaginal pH was alkaline, reduced ability to kill pathogens. Meanwhile, the vaginal mucosa atrophy, the Pifei thin, insufficient blood supply, so that the vagina lowered immunity, easy to breed bacteria invade cause inflammatory lesions. In addition, poor personal hygiene, nutritional deficiencies, especially B group vitamin deficiency may be related to pathogenesis.